csl antibody Search Results


rabbit polyclonal rbpj κ  (Novus Biologicals)
93
Novus Biologicals rabbit polyclonal rbpj κ
Rabbit Polyclonal Rbpj κ, supplied by Novus Biologicals, used in various techniques. Bioz Stars score: 93/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/result/rabbit polyclonal rbpj κ/product/Novus Biologicals
Average 93 stars, based on 1 article reviews
rabbit polyclonal rbpj κ - by Bioz Stars, 2026-03
93/100 stars
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rbpj  (Proteintech)
93
Proteintech rbpj
a Bioinformatic analysis of the potential binding proteins of the promoter <t>of</t> <t>DAPK3</t> via analysis the ChIP-seq datasets. b , c 786-O and ACHN cells were transfected with indicated plasmids for 24 h. Cells were harvested for western blotting analysis ( b ) and RT-qPCR analysis ( c ). Statistical significance was determined by one-way ANOVA followed by Tukey’s multiple comparisons test. Data presented as Mean ± SEM with three replicates ( n = 3). Ns not significant; ** P < 0.01; *** P < 0.001. d , e 786-O and ACHN cells were infected with indicated shRNAs for 72 h. Cells were harvested for western blotting analysis ( d ) and RT-qPCR analysis ( e ). Statistical significance was determined by one-way ANOVA followed by Tukey’s multiple comparisons test. Data presented as Mean ± SEM with three replicates ( n = 3). ** P < 0.01. f , g 786-O cells were treated with 0, 1 µM, 5 µM, and 10 µM the <t>RBPJ</t> inhibitors (the DAPK3 inhibitor) for 24 h. The WCL of cells were subjected to western blotting analysis ( f ) and RT-qPCR analysis ( g ). Statistical significance was determined by one-way ANOVA followed by Tukey’s multiple comparisons test. Data presented as Mean ± SEM with three replicates ( n = 3). Ns not significant; *** P < 0.001. h the ChIP-seq of RBPJ on the promoter region of DAPK3 . i 786-O cells were infected with indicated shRNAs for 72 h. Cells were harvested for ChIP-qPCR analysis by using the IgG or RBPJ antibodies. Statistical significance was determined by one-way ANOVA followed by Tukey’s multiple comparisons test. Data presented as Mean ± SEM with three replicates ( n = 3). ** P < 0.01; *** P < 0.001. j 786-O cells were transfected with indicated plasmids for 24 h. Cells were harvested for ChIP-qPCR analysis by using the IgG or RBPJ antibodies. Statistical significance was determined by one-way ANOVA followed by Tukey’s multiple comparisons test. Data presented as Mean ± SEM with three replicates ( n = 3). * P < 0.05; ** P < 0.01; *** P < 0.001. k The correlation between RBPJ and DAPK3 were analyzed by the GEPIA web tool ( http://gepia.cancer-pku.cn/ ) in different types of cancer. l 786-O cells were infected with indicated shRNAs for 72 h. Cells were harvested for western blotting analysis. m 786-O cells were infected with indicated shRNAs for 48 h. Then, cells were treated with or without the 5 µM RBPJ inhibitor for another 24 h. Cells were harvested for western blotting analysis.
Rbpj, supplied by Proteintech, used in various techniques. Bioz Stars score: 93/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/result/rbpj/product/Proteintech
Average 93 stars, based on 1 article reviews
rbpj - by Bioz Stars, 2026-03
93/100 stars
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panobacumab  (CSL Behring)
90
CSL Behring panobacumab
a Bioinformatic analysis of the potential binding proteins of the promoter <t>of</t> <t>DAPK3</t> via analysis the ChIP-seq datasets. b , c 786-O and ACHN cells were transfected with indicated plasmids for 24 h. Cells were harvested for western blotting analysis ( b ) and RT-qPCR analysis ( c ). Statistical significance was determined by one-way ANOVA followed by Tukey’s multiple comparisons test. Data presented as Mean ± SEM with three replicates ( n = 3). Ns not significant; ** P < 0.01; *** P < 0.001. d , e 786-O and ACHN cells were infected with indicated shRNAs for 72 h. Cells were harvested for western blotting analysis ( d ) and RT-qPCR analysis ( e ). Statistical significance was determined by one-way ANOVA followed by Tukey’s multiple comparisons test. Data presented as Mean ± SEM with three replicates ( n = 3). ** P < 0.01. f , g 786-O cells were treated with 0, 1 µM, 5 µM, and 10 µM the <t>RBPJ</t> inhibitors (the DAPK3 inhibitor) for 24 h. The WCL of cells were subjected to western blotting analysis ( f ) and RT-qPCR analysis ( g ). Statistical significance was determined by one-way ANOVA followed by Tukey’s multiple comparisons test. Data presented as Mean ± SEM with three replicates ( n = 3). Ns not significant; *** P < 0.001. h the ChIP-seq of RBPJ on the promoter region of DAPK3 . i 786-O cells were infected with indicated shRNAs for 72 h. Cells were harvested for ChIP-qPCR analysis by using the IgG or RBPJ antibodies. Statistical significance was determined by one-way ANOVA followed by Tukey’s multiple comparisons test. Data presented as Mean ± SEM with three replicates ( n = 3). ** P < 0.01; *** P < 0.001. j 786-O cells were transfected with indicated plasmids for 24 h. Cells were harvested for ChIP-qPCR analysis by using the IgG or RBPJ antibodies. Statistical significance was determined by one-way ANOVA followed by Tukey’s multiple comparisons test. Data presented as Mean ± SEM with three replicates ( n = 3). * P < 0.05; ** P < 0.01; *** P < 0.001. k The correlation between RBPJ and DAPK3 were analyzed by the GEPIA web tool ( http://gepia.cancer-pku.cn/ ) in different types of cancer. l 786-O cells were infected with indicated shRNAs for 72 h. Cells were harvested for western blotting analysis. m 786-O cells were infected with indicated shRNAs for 48 h. Then, cells were treated with or without the 5 µM RBPJ inhibitor for another 24 h. Cells were harvested for western blotting analysis.
Panobacumab, supplied by CSL Behring, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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Average 90 stars, based on 1 article reviews
panobacumab - by Bioz Stars, 2026-03
90/100 stars
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csl362  (CSL Limited)
90
CSL Limited csl362
a Bioinformatic analysis of the potential binding proteins of the promoter <t>of</t> <t>DAPK3</t> via analysis the ChIP-seq datasets. b , c 786-O and ACHN cells were transfected with indicated plasmids for 24 h. Cells were harvested for western blotting analysis ( b ) and RT-qPCR analysis ( c ). Statistical significance was determined by one-way ANOVA followed by Tukey’s multiple comparisons test. Data presented as Mean ± SEM with three replicates ( n = 3). Ns not significant; ** P < 0.01; *** P < 0.001. d , e 786-O and ACHN cells were infected with indicated shRNAs for 72 h. Cells were harvested for western blotting analysis ( d ) and RT-qPCR analysis ( e ). Statistical significance was determined by one-way ANOVA followed by Tukey’s multiple comparisons test. Data presented as Mean ± SEM with three replicates ( n = 3). ** P < 0.01. f , g 786-O cells were treated with 0, 1 µM, 5 µM, and 10 µM the <t>RBPJ</t> inhibitors (the DAPK3 inhibitor) for 24 h. The WCL of cells were subjected to western blotting analysis ( f ) and RT-qPCR analysis ( g ). Statistical significance was determined by one-way ANOVA followed by Tukey’s multiple comparisons test. Data presented as Mean ± SEM with three replicates ( n = 3). Ns not significant; *** P < 0.001. h the ChIP-seq of RBPJ on the promoter region of DAPK3 . i 786-O cells were infected with indicated shRNAs for 72 h. Cells were harvested for ChIP-qPCR analysis by using the IgG or RBPJ antibodies. Statistical significance was determined by one-way ANOVA followed by Tukey’s multiple comparisons test. Data presented as Mean ± SEM with three replicates ( n = 3). ** P < 0.01; *** P < 0.001. j 786-O cells were transfected with indicated plasmids for 24 h. Cells were harvested for ChIP-qPCR analysis by using the IgG or RBPJ antibodies. Statistical significance was determined by one-way ANOVA followed by Tukey’s multiple comparisons test. Data presented as Mean ± SEM with three replicates ( n = 3). * P < 0.05; ** P < 0.01; *** P < 0.001. k The correlation between RBPJ and DAPK3 were analyzed by the GEPIA web tool ( http://gepia.cancer-pku.cn/ ) in different types of cancer. l 786-O cells were infected with indicated shRNAs for 72 h. Cells were harvested for western blotting analysis. m 786-O cells were infected with indicated shRNAs for 48 h. Then, cells were treated with or without the 5 µM RBPJ inhibitor for another 24 h. Cells were harvested for western blotting analysis.
Csl362, supplied by CSL Limited, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/result/csl362/product/CSL Limited
Average 90 stars, based on 1 article reviews
csl362 - by Bioz Stars, 2026-03
90/100 stars
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purified hf.ix antibody  (CSL Behring)
90
CSL Behring purified hf.ix antibody
a Bioinformatic analysis of the potential binding proteins of the promoter <t>of</t> <t>DAPK3</t> via analysis the ChIP-seq datasets. b , c 786-O and ACHN cells were transfected with indicated plasmids for 24 h. Cells were harvested for western blotting analysis ( b ) and RT-qPCR analysis ( c ). Statistical significance was determined by one-way ANOVA followed by Tukey’s multiple comparisons test. Data presented as Mean ± SEM with three replicates ( n = 3). Ns not significant; ** P < 0.01; *** P < 0.001. d , e 786-O and ACHN cells were infected with indicated shRNAs for 72 h. Cells were harvested for western blotting analysis ( d ) and RT-qPCR analysis ( e ). Statistical significance was determined by one-way ANOVA followed by Tukey’s multiple comparisons test. Data presented as Mean ± SEM with three replicates ( n = 3). ** P < 0.01. f , g 786-O cells were treated with 0, 1 µM, 5 µM, and 10 µM the <t>RBPJ</t> inhibitors (the DAPK3 inhibitor) for 24 h. The WCL of cells were subjected to western blotting analysis ( f ) and RT-qPCR analysis ( g ). Statistical significance was determined by one-way ANOVA followed by Tukey’s multiple comparisons test. Data presented as Mean ± SEM with three replicates ( n = 3). Ns not significant; *** P < 0.001. h the ChIP-seq of RBPJ on the promoter region of DAPK3 . i 786-O cells were infected with indicated shRNAs for 72 h. Cells were harvested for ChIP-qPCR analysis by using the IgG or RBPJ antibodies. Statistical significance was determined by one-way ANOVA followed by Tukey’s multiple comparisons test. Data presented as Mean ± SEM with three replicates ( n = 3). ** P < 0.01; *** P < 0.001. j 786-O cells were transfected with indicated plasmids for 24 h. Cells were harvested for ChIP-qPCR analysis by using the IgG or RBPJ antibodies. Statistical significance was determined by one-way ANOVA followed by Tukey’s multiple comparisons test. Data presented as Mean ± SEM with three replicates ( n = 3). * P < 0.05; ** P < 0.01; *** P < 0.001. k The correlation between RBPJ and DAPK3 were analyzed by the GEPIA web tool ( http://gepia.cancer-pku.cn/ ) in different types of cancer. l 786-O cells were infected with indicated shRNAs for 72 h. Cells were harvested for western blotting analysis. m 786-O cells were infected with indicated shRNAs for 48 h. Then, cells were treated with or without the 5 µM RBPJ inhibitor for another 24 h. Cells were harvested for western blotting analysis.
Purified Hf.Ix Antibody, supplied by CSL Behring, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/result/purified hf.ix antibody/product/CSL Behring
Average 90 stars, based on 1 article reviews
purified hf.ix antibody - by Bioz Stars, 2026-03
90/100 stars
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ivig (privigen; 100 mg/ml)  (CSL Behring)
90
CSL Behring ivig (privigen; 100 mg/ml)
a Bioinformatic analysis of the potential binding proteins of the promoter <t>of</t> <t>DAPK3</t> via analysis the ChIP-seq datasets. b , c 786-O and ACHN cells were transfected with indicated plasmids for 24 h. Cells were harvested for western blotting analysis ( b ) and RT-qPCR analysis ( c ). Statistical significance was determined by one-way ANOVA followed by Tukey’s multiple comparisons test. Data presented as Mean ± SEM with three replicates ( n = 3). Ns not significant; ** P < 0.01; *** P < 0.001. d , e 786-O and ACHN cells were infected with indicated shRNAs for 72 h. Cells were harvested for western blotting analysis ( d ) and RT-qPCR analysis ( e ). Statistical significance was determined by one-way ANOVA followed by Tukey’s multiple comparisons test. Data presented as Mean ± SEM with three replicates ( n = 3). ** P < 0.01. f , g 786-O cells were treated with 0, 1 µM, 5 µM, and 10 µM the <t>RBPJ</t> inhibitors (the DAPK3 inhibitor) for 24 h. The WCL of cells were subjected to western blotting analysis ( f ) and RT-qPCR analysis ( g ). Statistical significance was determined by one-way ANOVA followed by Tukey’s multiple comparisons test. Data presented as Mean ± SEM with three replicates ( n = 3). Ns not significant; *** P < 0.001. h the ChIP-seq of RBPJ on the promoter region of DAPK3 . i 786-O cells were infected with indicated shRNAs for 72 h. Cells were harvested for ChIP-qPCR analysis by using the IgG or RBPJ antibodies. Statistical significance was determined by one-way ANOVA followed by Tukey’s multiple comparisons test. Data presented as Mean ± SEM with three replicates ( n = 3). ** P < 0.01; *** P < 0.001. j 786-O cells were transfected with indicated plasmids for 24 h. Cells were harvested for ChIP-qPCR analysis by using the IgG or RBPJ antibodies. Statistical significance was determined by one-way ANOVA followed by Tukey’s multiple comparisons test. Data presented as Mean ± SEM with three replicates ( n = 3). * P < 0.05; ** P < 0.01; *** P < 0.001. k The correlation between RBPJ and DAPK3 were analyzed by the GEPIA web tool ( http://gepia.cancer-pku.cn/ ) in different types of cancer. l 786-O cells were infected with indicated shRNAs for 72 h. Cells were harvested for western blotting analysis. m 786-O cells were infected with indicated shRNAs for 48 h. Then, cells were treated with or without the 5 µM RBPJ inhibitor for another 24 h. Cells were harvested for western blotting analysis.
Ivig (Privigen; 100 Mg/Ml), supplied by CSL Behring, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/result/ivig (privigen; 100 mg/ml)/product/CSL Behring
Average 90 stars, based on 1 article reviews
ivig (privigen; 100 mg/ml) - by Bioz Stars, 2026-03
90/100 stars
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hsv-1 neutralizing antibodies  (CSL Behring)
90
CSL Behring hsv-1 neutralizing antibodies
a Bioinformatic analysis of the potential binding proteins of the promoter <t>of</t> <t>DAPK3</t> via analysis the ChIP-seq datasets. b , c 786-O and ACHN cells were transfected with indicated plasmids for 24 h. Cells were harvested for western blotting analysis ( b ) and RT-qPCR analysis ( c ). Statistical significance was determined by one-way ANOVA followed by Tukey’s multiple comparisons test. Data presented as Mean ± SEM with three replicates ( n = 3). Ns not significant; ** P < 0.01; *** P < 0.001. d , e 786-O and ACHN cells were infected with indicated shRNAs for 72 h. Cells were harvested for western blotting analysis ( d ) and RT-qPCR analysis ( e ). Statistical significance was determined by one-way ANOVA followed by Tukey’s multiple comparisons test. Data presented as Mean ± SEM with three replicates ( n = 3). ** P < 0.01. f , g 786-O cells were treated with 0, 1 µM, 5 µM, and 10 µM the <t>RBPJ</t> inhibitors (the DAPK3 inhibitor) for 24 h. The WCL of cells were subjected to western blotting analysis ( f ) and RT-qPCR analysis ( g ). Statistical significance was determined by one-way ANOVA followed by Tukey’s multiple comparisons test. Data presented as Mean ± SEM with three replicates ( n = 3). Ns not significant; *** P < 0.001. h the ChIP-seq of RBPJ on the promoter region of DAPK3 . i 786-O cells were infected with indicated shRNAs for 72 h. Cells were harvested for ChIP-qPCR analysis by using the IgG or RBPJ antibodies. Statistical significance was determined by one-way ANOVA followed by Tukey’s multiple comparisons test. Data presented as Mean ± SEM with three replicates ( n = 3). ** P < 0.01; *** P < 0.001. j 786-O cells were transfected with indicated plasmids for 24 h. Cells were harvested for ChIP-qPCR analysis by using the IgG or RBPJ antibodies. Statistical significance was determined by one-way ANOVA followed by Tukey’s multiple comparisons test. Data presented as Mean ± SEM with three replicates ( n = 3). * P < 0.05; ** P < 0.01; *** P < 0.001. k The correlation between RBPJ and DAPK3 were analyzed by the GEPIA web tool ( http://gepia.cancer-pku.cn/ ) in different types of cancer. l 786-O cells were infected with indicated shRNAs for 72 h. Cells were harvested for western blotting analysis. m 786-O cells were infected with indicated shRNAs for 48 h. Then, cells were treated with or without the 5 µM RBPJ inhibitor for another 24 h. Cells were harvested for western blotting analysis.
Hsv 1 Neutralizing Antibodies, supplied by CSL Behring, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/result/hsv-1 neutralizing antibodies/product/CSL Behring
Average 90 stars, based on 1 article reviews
hsv-1 neutralizing antibodies - by Bioz Stars, 2026-03
90/100 stars
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beriglobin antibody  (CSL Behring)
90
CSL Behring beriglobin antibody
a Bioinformatic analysis of the potential binding proteins of the promoter <t>of</t> <t>DAPK3</t> via analysis the ChIP-seq datasets. b , c 786-O and ACHN cells were transfected with indicated plasmids for 24 h. Cells were harvested for western blotting analysis ( b ) and RT-qPCR analysis ( c ). Statistical significance was determined by one-way ANOVA followed by Tukey’s multiple comparisons test. Data presented as Mean ± SEM with three replicates ( n = 3). Ns not significant; ** P < 0.01; *** P < 0.001. d , e 786-O and ACHN cells were infected with indicated shRNAs for 72 h. Cells were harvested for western blotting analysis ( d ) and RT-qPCR analysis ( e ). Statistical significance was determined by one-way ANOVA followed by Tukey’s multiple comparisons test. Data presented as Mean ± SEM with three replicates ( n = 3). ** P < 0.01. f , g 786-O cells were treated with 0, 1 µM, 5 µM, and 10 µM the <t>RBPJ</t> inhibitors (the DAPK3 inhibitor) for 24 h. The WCL of cells were subjected to western blotting analysis ( f ) and RT-qPCR analysis ( g ). Statistical significance was determined by one-way ANOVA followed by Tukey’s multiple comparisons test. Data presented as Mean ± SEM with three replicates ( n = 3). Ns not significant; *** P < 0.001. h the ChIP-seq of RBPJ on the promoter region of DAPK3 . i 786-O cells were infected with indicated shRNAs for 72 h. Cells were harvested for ChIP-qPCR analysis by using the IgG or RBPJ antibodies. Statistical significance was determined by one-way ANOVA followed by Tukey’s multiple comparisons test. Data presented as Mean ± SEM with three replicates ( n = 3). ** P < 0.01; *** P < 0.001. j 786-O cells were transfected with indicated plasmids for 24 h. Cells were harvested for ChIP-qPCR analysis by using the IgG or RBPJ antibodies. Statistical significance was determined by one-way ANOVA followed by Tukey’s multiple comparisons test. Data presented as Mean ± SEM with three replicates ( n = 3). * P < 0.05; ** P < 0.01; *** P < 0.001. k The correlation between RBPJ and DAPK3 were analyzed by the GEPIA web tool ( http://gepia.cancer-pku.cn/ ) in different types of cancer. l 786-O cells were infected with indicated shRNAs for 72 h. Cells were harvested for western blotting analysis. m 786-O cells were infected with indicated shRNAs for 48 h. Then, cells were treated with or without the 5 µM RBPJ inhibitor for another 24 h. Cells were harvested for western blotting analysis.
Beriglobin Antibody, supplied by CSL Behring, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/result/beriglobin antibody/product/CSL Behring
Average 90 stars, based on 1 article reviews
beriglobin antibody - by Bioz Stars, 2026-03
90/100 stars
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anti-cd123 antibody or fragment thereof  (CSL Limited)
90
CSL Limited anti-cd123 antibody or fragment thereof
a Bioinformatic analysis of the potential binding proteins of the promoter <t>of</t> <t>DAPK3</t> via analysis the ChIP-seq datasets. b , c 786-O and ACHN cells were transfected with indicated plasmids for 24 h. Cells were harvested for western blotting analysis ( b ) and RT-qPCR analysis ( c ). Statistical significance was determined by one-way ANOVA followed by Tukey’s multiple comparisons test. Data presented as Mean ± SEM with three replicates ( n = 3). Ns not significant; ** P < 0.01; *** P < 0.001. d , e 786-O and ACHN cells were infected with indicated shRNAs for 72 h. Cells were harvested for western blotting analysis ( d ) and RT-qPCR analysis ( e ). Statistical significance was determined by one-way ANOVA followed by Tukey’s multiple comparisons test. Data presented as Mean ± SEM with three replicates ( n = 3). ** P < 0.01. f , g 786-O cells were treated with 0, 1 µM, 5 µM, and 10 µM the <t>RBPJ</t> inhibitors (the DAPK3 inhibitor) for 24 h. The WCL of cells were subjected to western blotting analysis ( f ) and RT-qPCR analysis ( g ). Statistical significance was determined by one-way ANOVA followed by Tukey’s multiple comparisons test. Data presented as Mean ± SEM with three replicates ( n = 3). Ns not significant; *** P < 0.001. h the ChIP-seq of RBPJ on the promoter region of DAPK3 . i 786-O cells were infected with indicated shRNAs for 72 h. Cells were harvested for ChIP-qPCR analysis by using the IgG or RBPJ antibodies. Statistical significance was determined by one-way ANOVA followed by Tukey’s multiple comparisons test. Data presented as Mean ± SEM with three replicates ( n = 3). ** P < 0.01; *** P < 0.001. j 786-O cells were transfected with indicated plasmids for 24 h. Cells were harvested for ChIP-qPCR analysis by using the IgG or RBPJ antibodies. Statistical significance was determined by one-way ANOVA followed by Tukey’s multiple comparisons test. Data presented as Mean ± SEM with three replicates ( n = 3). * P < 0.05; ** P < 0.01; *** P < 0.001. k The correlation between RBPJ and DAPK3 were analyzed by the GEPIA web tool ( http://gepia.cancer-pku.cn/ ) in different types of cancer. l 786-O cells were infected with indicated shRNAs for 72 h. Cells were harvested for western blotting analysis. m 786-O cells were infected with indicated shRNAs for 48 h. Then, cells were treated with or without the 5 µM RBPJ inhibitor for another 24 h. Cells were harvested for western blotting analysis.
Anti Cd123 Antibody Or Fragment Thereof, supplied by CSL Limited, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/result/anti-cd123 antibody or fragment thereof/product/CSL Limited
Average 90 stars, based on 1 article reviews
anti-cd123 antibody or fragment thereof - by Bioz Stars, 2026-03
90/100 stars
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polyclonal cmv igg  (CSL Behring)
90
CSL Behring polyclonal cmv igg
gB-specific mAbs mediate negligible NK activation ADCC responses when compared to <t>polyclonal</t> <t>CMV-specific</t> antibody preparation (Cytogam). gB-specific mAbs were screened for mediation of NK cell (A) CD107a degranulation and (B) CD16 downregulation (DRI) as measures of antibody-mediated NK cell activation. Data are presented as mAb specific response against AD169r-GFP infected ARPE19 cells minus mock infected negative control cells.
Polyclonal Cmv Igg, supplied by CSL Behring, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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Average 90 stars, based on 1 article reviews
polyclonal cmv igg - by Bioz Stars, 2026-03
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idarucizumab antibody  (CSL Behring)
90
CSL Behring idarucizumab antibody
gB-specific mAbs mediate negligible NK activation ADCC responses when compared to <t>polyclonal</t> <t>CMV-specific</t> antibody preparation (Cytogam). gB-specific mAbs were screened for mediation of NK cell (A) CD107a degranulation and (B) CD16 downregulation (DRI) as measures of antibody-mediated NK cell activation. Data are presented as mAb specific response against AD169r-GFP infected ARPE19 cells minus mock infected negative control cells.
Idarucizumab Antibody, supplied by CSL Behring, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/result/idarucizumab antibody/product/CSL Behring
Average 90 stars, based on 1 article reviews
idarucizumab antibody - by Bioz Stars, 2026-03
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fxiia inhibitory antibody (3f7  (CSL Limited)
90
CSL Limited fxiia inhibitory antibody (3f7
gB-specific mAbs mediate negligible NK activation ADCC responses when compared to <t>polyclonal</t> <t>CMV-specific</t> antibody preparation (Cytogam). gB-specific mAbs were screened for mediation of NK cell (A) CD107a degranulation and (B) CD16 downregulation (DRI) as measures of antibody-mediated NK cell activation. Data are presented as mAb specific response against AD169r-GFP infected ARPE19 cells minus mock infected negative control cells.
Fxiia Inhibitory Antibody (3f7, supplied by CSL Limited, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/result/fxiia inhibitory antibody (3f7/product/CSL Limited
Average 90 stars, based on 1 article reviews
fxiia inhibitory antibody (3f7 - by Bioz Stars, 2026-03
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Image Search Results


a Bioinformatic analysis of the potential binding proteins of the promoter of DAPK3 via analysis the ChIP-seq datasets. b , c 786-O and ACHN cells were transfected with indicated plasmids for 24 h. Cells were harvested for western blotting analysis ( b ) and RT-qPCR analysis ( c ). Statistical significance was determined by one-way ANOVA followed by Tukey’s multiple comparisons test. Data presented as Mean ± SEM with three replicates ( n = 3). Ns not significant; ** P < 0.01; *** P < 0.001. d , e 786-O and ACHN cells were infected with indicated shRNAs for 72 h. Cells were harvested for western blotting analysis ( d ) and RT-qPCR analysis ( e ). Statistical significance was determined by one-way ANOVA followed by Tukey’s multiple comparisons test. Data presented as Mean ± SEM with three replicates ( n = 3). ** P < 0.01. f , g 786-O cells were treated with 0, 1 µM, 5 µM, and 10 µM the RBPJ inhibitors (the DAPK3 inhibitor) for 24 h. The WCL of cells were subjected to western blotting analysis ( f ) and RT-qPCR analysis ( g ). Statistical significance was determined by one-way ANOVA followed by Tukey’s multiple comparisons test. Data presented as Mean ± SEM with three replicates ( n = 3). Ns not significant; *** P < 0.001. h the ChIP-seq of RBPJ on the promoter region of DAPK3 . i 786-O cells were infected with indicated shRNAs for 72 h. Cells were harvested for ChIP-qPCR analysis by using the IgG or RBPJ antibodies. Statistical significance was determined by one-way ANOVA followed by Tukey’s multiple comparisons test. Data presented as Mean ± SEM with three replicates ( n = 3). ** P < 0.01; *** P < 0.001. j 786-O cells were transfected with indicated plasmids for 24 h. Cells were harvested for ChIP-qPCR analysis by using the IgG or RBPJ antibodies. Statistical significance was determined by one-way ANOVA followed by Tukey’s multiple comparisons test. Data presented as Mean ± SEM with three replicates ( n = 3). * P < 0.05; ** P < 0.01; *** P < 0.001. k The correlation between RBPJ and DAPK3 were analyzed by the GEPIA web tool ( http://gepia.cancer-pku.cn/ ) in different types of cancer. l 786-O cells were infected with indicated shRNAs for 72 h. Cells were harvested for western blotting analysis. m 786-O cells were infected with indicated shRNAs for 48 h. Then, cells were treated with or without the 5 µM RBPJ inhibitor for another 24 h. Cells were harvested for western blotting analysis.

Journal: Cell Death & Disease

Article Title: The RBPJ/DAPK3/UBE3A signaling axis induces PBRM1 degradation to modulate the sensitivity of renal cell carcinoma to CDK4/6 inhibitors

doi: 10.1038/s41419-022-04760-6

Figure Lengend Snippet: a Bioinformatic analysis of the potential binding proteins of the promoter of DAPK3 via analysis the ChIP-seq datasets. b , c 786-O and ACHN cells were transfected with indicated plasmids for 24 h. Cells were harvested for western blotting analysis ( b ) and RT-qPCR analysis ( c ). Statistical significance was determined by one-way ANOVA followed by Tukey’s multiple comparisons test. Data presented as Mean ± SEM with three replicates ( n = 3). Ns not significant; ** P < 0.01; *** P < 0.001. d , e 786-O and ACHN cells were infected with indicated shRNAs for 72 h. Cells were harvested for western blotting analysis ( d ) and RT-qPCR analysis ( e ). Statistical significance was determined by one-way ANOVA followed by Tukey’s multiple comparisons test. Data presented as Mean ± SEM with three replicates ( n = 3). ** P < 0.01. f , g 786-O cells were treated with 0, 1 µM, 5 µM, and 10 µM the RBPJ inhibitors (the DAPK3 inhibitor) for 24 h. The WCL of cells were subjected to western blotting analysis ( f ) and RT-qPCR analysis ( g ). Statistical significance was determined by one-way ANOVA followed by Tukey’s multiple comparisons test. Data presented as Mean ± SEM with three replicates ( n = 3). Ns not significant; *** P < 0.001. h the ChIP-seq of RBPJ on the promoter region of DAPK3 . i 786-O cells were infected with indicated shRNAs for 72 h. Cells were harvested for ChIP-qPCR analysis by using the IgG or RBPJ antibodies. Statistical significance was determined by one-way ANOVA followed by Tukey’s multiple comparisons test. Data presented as Mean ± SEM with three replicates ( n = 3). ** P < 0.01; *** P < 0.001. j 786-O cells were transfected with indicated plasmids for 24 h. Cells were harvested for ChIP-qPCR analysis by using the IgG or RBPJ antibodies. Statistical significance was determined by one-way ANOVA followed by Tukey’s multiple comparisons test. Data presented as Mean ± SEM with three replicates ( n = 3). * P < 0.05; ** P < 0.01; *** P < 0.001. k The correlation between RBPJ and DAPK3 were analyzed by the GEPIA web tool ( http://gepia.cancer-pku.cn/ ) in different types of cancer. l 786-O cells were infected with indicated shRNAs for 72 h. Cells were harvested for western blotting analysis. m 786-O cells were infected with indicated shRNAs for 48 h. Then, cells were treated with or without the 5 µM RBPJ inhibitor for another 24 h. Cells were harvested for western blotting analysis.

Article Snippet: The antibodies used as follows: UBE3A (10344-1-AP, Proteintech; 1:1000 dilution), PBRM1 (12563-1-AP, Proteintech; 1:500 dilution), DAPK3 (2928, Cell signaling technology, 1:1000 dilution), RBPJ (14613-1-AP, Proteintech; 1:1000 dilution); P21 (10355-1-AP, Proteintech; 1:1000 dilution); GAPDH (10494-1-AP, Proteintech; 1:10000 dilution).

Techniques: Binding Assay, ChIP-sequencing, Transfection, Western Blot, Quantitative RT-PCR, Infection, ChIP-qPCR

DAPK3 competed with PKA to bind with UBE3A and enhance the PBRM1 degradation in renal cancer cells. PBPJ transcriptionally regulated DAPK3 expression and then promoted UBE3A-mediated degradation of PBRM1. Then, PBRM1 increased the p21 expression and sensitized renal cancer cells to CDK4/6 inhibitors. In combination with RBPJ inhibitors, CDK4/6 inhibitors synergistically enhanced renal cancer cells.

Journal: Cell Death & Disease

Article Title: The RBPJ/DAPK3/UBE3A signaling axis induces PBRM1 degradation to modulate the sensitivity of renal cell carcinoma to CDK4/6 inhibitors

doi: 10.1038/s41419-022-04760-6

Figure Lengend Snippet: DAPK3 competed with PKA to bind with UBE3A and enhance the PBRM1 degradation in renal cancer cells. PBPJ transcriptionally regulated DAPK3 expression and then promoted UBE3A-mediated degradation of PBRM1. Then, PBRM1 increased the p21 expression and sensitized renal cancer cells to CDK4/6 inhibitors. In combination with RBPJ inhibitors, CDK4/6 inhibitors synergistically enhanced renal cancer cells.

Article Snippet: The antibodies used as follows: UBE3A (10344-1-AP, Proteintech; 1:1000 dilution), PBRM1 (12563-1-AP, Proteintech; 1:500 dilution), DAPK3 (2928, Cell signaling technology, 1:1000 dilution), RBPJ (14613-1-AP, Proteintech; 1:1000 dilution); P21 (10355-1-AP, Proteintech; 1:1000 dilution); GAPDH (10494-1-AP, Proteintech; 1:10000 dilution).

Techniques: Expressing

gB-specific mAbs mediate negligible NK activation ADCC responses when compared to polyclonal CMV-specific antibody preparation (Cytogam). gB-specific mAbs were screened for mediation of NK cell (A) CD107a degranulation and (B) CD16 downregulation (DRI) as measures of antibody-mediated NK cell activation. Data are presented as mAb specific response against AD169r-GFP infected ARPE19 cells minus mock infected negative control cells.

Journal: bioRxiv

Article Title: Specificity and effector functions of non-neutralizing gB-specific monoclonal antibodies isolated from healthy individuals with human cytomegalovirus infection

doi: 10.1101/2020.01.14.907204

Figure Lengend Snippet: gB-specific mAbs mediate negligible NK activation ADCC responses when compared to polyclonal CMV-specific antibody preparation (Cytogam). gB-specific mAbs were screened for mediation of NK cell (A) CD107a degranulation and (B) CD16 downregulation (DRI) as measures of antibody-mediated NK cell activation. Data are presented as mAb specific response against AD169r-GFP infected ARPE19 cells minus mock infected negative control cells.

Article Snippet: Purity and identity were confirmed by Western blot using polyclonal CMV IgG (Cytogam – CSL Behring) or monoclonal antibodies SM10 (Domain I) and SM5-1 (Domain II).

Techniques: Activation Assay, Infection, Negative Control